References

Bouza E, Burillo A, Muñoz P Catheter-related infections; diagnosis and intravascular treatment.. Clin Microbiol Infect. 2002; 8:(5)265-274 https://doi.org/10.1046/j.1469-0691.2002.00385.x

Clayton W, Mah TF Molecular mechanisms of biofilm-based antibiotic resistance and tolerance in pathogenic bacteria.. FEMS Microbiology Reviews. 2017; 41:(3)276-301 https://doi.org/10.1093/femsre/fux010

Fibrinogen: nature's duct tape! Transverse covalent bonds create a stretchy clot. SMART Team poster presentation.. 2012. http://tinyurl.com/y29d2tbf

Keller JE, Hindman JW, Mehall JR, Smith SD Enoxaparin inhibits fibrin sheath formation and decreases central venous colonization following bacteremic challenge.. Crit Care Med. 2006; 34:(5)1450-1455 https://doi.org/10.1097/01.CCM.0000215832.40827.71

Kendall K, Roberts A Van der Waals forces influencing adhesion of cells.. Philos Trans R Soc Lond B Biol Sci. 2015; 370:(1661) https://doi.org/10.1098/rstb.2014.0078

Krausz DJ, Fisher JS, Rosen G Retained fibrin sheaths: chest computed tomography findings and clinical associations.. J Thorac Imaging. 2014; 29:(2)118-24 https://doi.org/10.1097/RTI.0b013e318299ff22

Kumwenda M, Dougherty L, Spooner H, Jackson V, Mitra S, Inston N Managing dysfunctional central venous access devices: a practical approach to urokinase thrombolysis.. Br J Nurs.. 2018a; 27:(2)S4-S10

Kuter DJ Thrombotic complication of central venous catheters in cancer patients.. Oncologist. 2004; 9:(2)207-216 https://doi.org/10.1634/theoncologist.9-2-207

Mayo DJ Fibrin sheath formation and chemotherapy extravasation: a case report.. Support Care Cancer. 1998; 6:(1)51-56 https://doi.org/10.1007/s005200050132

Mehall JR, Saltzman DA, Jackson RJ, Smith SD Fibrin sheath enhances central venous catheter infection.. Crit Care Med. 2002; 30:(4)908-912

Motin J, Fischer G, Evreux J Intérêt de la voie sous-claviculaire en réanimation prolongée (Importance of the subclavicular route in prolonged resuscitation).. Lyon Med. 1964; 212:583-593

Niehaus W: University of Southampton; 2015 https://eprints.soton.ac.uk/382952/

Nycz M, Paradowska E, Arkusz K, Kudliński B, Krasicka-Cydzik E Surface analysis of long-term hemodialysis catheters made of carbothane (poly(carbonate)urethane) before and after implantation in the patients' bodies.. Acta Bioeng Biomech. 2018; 20:(2)47-53

Oliveira BL, Caravan P Peptide-based fibrin-targeting probes for thrombus imaging.. Dalton Trans. 2017; 46:(42)14488-14508 https://doi.org/10.1039/c7dt02634j

Pascual A Pathogenesis of catheter-related infections: lessons for new designs.. Clin Microbiol Infect. 2002; 8:(5)256-264 https://doi.org/10.1046/j.1469-0691.2002.00418.x

Passos da Silva D, Schofield MC, Parsek MR, Tseng BS An update on socialmicrobiology of quorum sensing in gram negative bacteria.. Pathogens. 2017; 6:(4) https://doi.org/10.3390/pathogens6040051

Reddy AS, Lang EV, Cutts J, Loh S, Rosen MP Fibrin sheath removal from central venous catheters: an internal snare manoeuvre.. Nephrol Dial Transplant. 2007; 22:(6)1762-1765

Ryder M The role of biofilm in vascular catheter-related infections.. New Developments in Vascular Disease. 2001; 2:15-25

Seifert H, Jansen B, Farr B, 1st edn.. London: Marcel Dekker; 1997

Fibrin sheath-associated endovascular infection of the heart: the Trojan horse of indwelling central venous catheters.. 2017. https://doi.org/10.1136/bcr-2016-219060

Sylvia CJ, Wagel MA, Giare-Patel K, Spangler TA, Breznock EM, Gupta N Chlorhexidine-coated peripherally inserted central catheters reduce fibroblastic sleeve formation in an in vivo ovine model.. J Vasc Access. 2018; 19:(6)644-650 https://doi.org/10.1177/1129729818769033

Tang S, Beigel R, Arsanjani R, Larson B, Luthringer D, Siegel R Infective endovascular fibrin sheath vegetations—a new cause of bacteremia detected by transesophageal echocardiogram.. Am J Med. 2015; 128:(9)1029-1038 https://doi.org/10.1016/j.amjmed.2015.03.019

Wallace A, Albadawi H, Patel N Anti-fouling strategies for central venous catheters.. Cardiovasc Diagn Ther. 2017; 7:(Suppl 3)S246-S257 https://doi.org/10.21037/cdt.2017.09.18

Wang H, Tong H, Liu H Effectiveness of antimicrobial-coated central venous catheters for preventing catheter related blood stream infections with the implementation of bundles; a systematic review and network meta-analysis.. Ann Intensive Care. 2018; 8:(1) https://doi.org/10.1186/s13613-018-0416-4

Xiang DZ, Verbeken EK, Van Lommel AT, Stas M, De Wever I Composition and formation of the sleeve enveloping a central venous catheter.. J Vasc Surg. 1998; 28:(2)260-271

Central venous access device-related sheaths: a predictor of infective and thrombotic incidence?

24 October 2019
Volume 28 · Issue 19

Abstract

Central vascular access device (CVAD)-related sheaths, sometimes described as ‘fibrin sheaths’, may result in minor or significant sequelae, from persistent withdrawal occlusion (PWO) to infective sheaths associated with increased morbidity and mortality. The authors studied 179 patients who underwent isotope scans, where isotope was infused via the CVAD. Isotope was found to bind to the sheaths around the catheters of some patients. The amount of uptake was taken to be an extent to which a sheath had developed around the CVAD. The degree of uptake of isotope was categorised into three groups: low uptake, moderate uptake and high uptake. Patients were then followed up from the date the CVAD was inserted to 12 months after the date of the isotope scan, until the device was removed or to the date the patient died, to identify incidence of infection, thrombosis and PWO. PWO incidence in all levels of uptake was around 5–7%. Bloodstream infection (BSI) incidence for low uptake was 7% (9/130), moderate uptake 10% (3/30) and for patients with significant uptake 16% (3/19). Thrombosis for no uptake was less than 1% (1/130), moderate uptake 7% (2/30), and significant uptake had no incidence of thrombosis. Total complications: no uptake 15%, moderate uptake 23% and significant uptake 21%. This single-centre study showed that patients with isotope-highlighted sheaths experienced higher incidence of infective, thrombotic and total complications.

Central venous access device (CVAD)-related sheaths are a phenomenon that may lead to minor functional problems of CVADs or significant sequelae for patients, such as the development of extravasation (Mayo, 1998; Kumwenda et al, 2018a) and are associated with venous occlusion (Krausz et al, 2014). CVAD functionality may be adversely affected—there may be failure to withdraw blood (persistent withdrawal occlusion), or infusion rates can be reduced (Kuter, 2004). Both of these lead to delayed or missed intravenous therapy. Protocols within the authors’ institution for persistent withdrawal occlusion related to CVAD sheaths may include chest X-ray, administration of thrombolytic agents and saline infusions to ensure the device is safe to use. This process has a negative impact on the patient experience and clinical and financial resources.

The incidence of CVAD sheaths has been reported to be 78-100% (Kuter, 2004). Seifert et al (1997) explained that virtually all foreign-body material soon becomes coated with glycoproteins containing fibrinogen, fibronectin, collagen and other proteins.

Register now to continue reading

Thank you for visiting British Journal of Nursing and reading some of our peer-reviewed resources for nurses. To read more, please register today. You’ll enjoy the following great benefits:

What's included

  • Limited access to clinical or professional articles

  • Unlimited access to the latest news, blogs and video content